Teratogen Endotoxin in every vial of mRNA Jab

We know that the mRNA jabs have caused Millions of people to suffer. Now look at what we can expect it will do to Boys and Girls born to jabbed Mothers.

As I briefly mentioned in a previous article¹, few people are aware that Pfizer has an ongoing Teratology study to see what effects their Covid19 jabs have on surviving Foetuses of Mothers who are jabbed before or during Pregnancy.

Teratology is the scientific study of Congenital Abnormalities and Abnormal Formations.

In this article I will cover the effects of the Endotoxin in every vial, arising from the use of E coli Bacteria in production of the mRNA jabs, ignoring the possible effects of the mRNA, impurity Plasmid DNA and other ingredients such as the 4 components of the Lipid Nanoparticles used to deliver the mRNA. There is an interesting paper on the Synergistic Toxicity of Endotoxin and synthetic or natural Covid19 Spike Protein on disease progression.²

Endotoxin Gelding of the Father

Of course Teratogenic effects of the jabs should look at the Father as well as the Mother, but Pfizer would not want to go down the path! So far I have found 2 cases of Hypogonadism listed as Adverse Events during the Pfizer jab trial.

GELDING is an appropriate term used for Endotoxin reduction of Testosterone production by Testes, both by direct inhibition of Leydig cell steroidogenic pathways and indirectly by reducing Pituitary Luteinizing Hormone drive, leading to a decline in Sperm production.³

I reported that Endotoxin in the jabs causes Heart damage via upregulation of miR-155 and now we see it is associated with decreased Male Fertility. So will those boys who suffered Myocarditis be less likely to procreate?

I asked Elon Musk, who recently told over 31 Million followers that

“To heir is human, To procreate is divine”,

if he knew Endotoxin in mRNA jabs is Teratogenic.

Endotoxin effects on the Mother

Recently I reported that Endotoxin depletes the Mother’s precious Egg Supply.⁴

As shown in the scheme below, both Mother and Foetus are threatened by Endotoxin circulating in their systems.⁵

This scheme demonstrates that Interleukin 1-beta, IL-1β is the “apex cytokine” in the inflammatory cascade of preterm birth and foetal inflammatory injury.

PR-A/PR-B = P4 Receptors A and B; PGs = Prostaglandins; MMPs = Matrix MetalloProteinases; PGF2α = Prostaglandin F2α; PLV = Periventricular Leukomalacia; BPD = BronchoPulmonary Dysplasia; NEC = Necrotizing EnteroColitis. Increasing color intensity represents increasing inflammatory response.

I have written about Pregnancies of jabbed Mothers that result in loss of the developing Child.⁶

Endotoxin Depletes the Child’s Egg Reserve

Teratology of Endotoxin has been shown to reduce the Fertility of the Female Offspring of experimental animals, as shown by a study from Israel. Pregnant Rats hit with intraperitoneal Endotoxin injections produced Intrauterine Growth Restriction and significantly lower levels of serum Anti-Müllerian Hormone in offspring.⁷

Here we see slices of the Ovaries of Rat Baby Pups demonstrating less Preantral Follicles in those whose Mothers were jabbed with Endotoxin. This correlated with increased Caspase-3 which is a Cysteine–aspartic acid protease that cleaves cellular targets and executes cell death.⁸

Caspase-3 may play a role in embryonic and hematopoietic stem cell differentiation, and its dysfunction could possibly lead to Malformations of the Foetus.⁹

Pfizer Teratology Trial

Study C4591022, entitled “Pfizer-BioNTech COVID-19 Vaccine Exposure during Pregnancy: A Non-Interventional Post-Approval Safety Study of Pregnancy and Infant Outcomes in the Organization of Teratology Information Specialists (OTIS)/MotherToBaby Pregnancy Registry.”

Final Protocol Submission: July 1, 2021

Study Completion: June 30, 2025

Final Report Submission: December 31, 2025

Because this study in Non-Interventional it is not classified technically as part of the Pfizer trials.¹⁰

Pfizer Post-Marketing Teratology reports

To April 2022, Pfizer reported 376 different types of “Congenital, familial and genetic disorders”, so I would soon go past email length if I tried to list them all.

Here are just a few with case numbers:

Congenital anomaly 46; Hypertrophic cardiomyopathy 40; Heart disease congenital 39; Atrial septal defect 38; Macroglossia 26; Foetal malformation 20; Ehlers-Danlos syndrome 18; Gilbert’s syndrome 17; Cerebral palsy 15; Factor V Leiden mutation 15; Ventricular septal defect 14; Trisomy 21 12; Hereditary angioedema 11; Hydrocele 11; Haemophilia 9; Huntington’s disease 9; Benign familial pemphigus 8; Factor VIII deficiency 8; Gene mutation 8; Anencephaly 7; Arnold-Chiari malformation 7; Arteriovenous malformation 7; Limb malformation 6; Spine malformation 5; Cerebrovascular arteriovenous malformation 4; Kidney malformation 4; Brain malformation 2; Skull malformation 2.

Endotoxin causes Foetal Heart Disease

“Cardia bifida constitutes such a cardiac malformation incompatible with early life and is best described by a split heart, the formation of two cardiac cylinders rather than one.”

Endotoxin is known to cause Cardia bifida.¹¹ Studies in Mice and Chick Embryos using Gene Transfection and tissue transplantation helped to elucidate the probable mechanisms. Endotoxin restricts cell migration of cardiac progenitors during the early stages of embryo development. Expressions of Nkx2.5 and GATA5 genes, associated with cardiomyocyte differentiation when heart tube fusion occurs, were disturbed. Cell proliferation decreased and cell apoptosis increased in the heart tube of chick embryos, correlated with pHIS3 and C-caspase3 immunohistological staining. Reactive Oxygen Species also contributed to the damage.

Looks like another article will be needed to explore the first generation Teratogenic impact of Endotoxin in mRNA jabs, and it will take perhaps 20 years to learn what will happen in the second generation descendants of Jabbees.

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1.

2. Petrlova J, et al. 2022. SARS-CoV-2 spike protein aggregation is triggered by bacterial lipopolysaccharide. https://febs.onlinelibrary.wiley.com/doi/epdf/10.1002/1873-3468.14490

3. Tremellen K. 2016. Gut Endotoxin Leading to a Decline IN Gonadal function (GELDING) - a novel theory for the development of late onset hypogonadism in obese men. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4918028/

4.

5. Coler BS, et al. 2021. Landscape of Preterm Birth Therapeutics and a Path Forward. https://www.mdpi.com/2077-0383/10/13/2912

6.

7. Shalom-Paz E, et al. 2017. IUGR induced by maternal chronic inflammation: long-term effect on offspring's ovaries in rat model-a preliminary report. https://link.springer.com/article/10.1007/s40618-017-0681-3

8. https://en.wikipedia.org/wiki/Caspase_3

9. https://www.cell.com/cell-stem-cell/fulltext/S1934-5909(08)00227-0

10. https://mothertobaby.org/ongoing-study/covid19-vaccines/

11. Jing Zhang, Guang Wang, Jia Liu, Lin-rui Gao, Meng Liu, Chao-jie Wang, Manli Chuai, Yongping Bao, Ge Li, Rui-man Li, Yu Zhang, Xuesong Yang. 2017. Gut microbiota-derived endotoxin enhanced the incidence of cardia bifida during cardiogenesis. https://onlinelibrary.wiley.com/doi/10.1002/jcp.26175