Risk factors for Stroke after mRNA Jabs point to Endotoxin
Pfizer reported thousands of cases of Stroke after its mRNA under 22 different headings in its reports. Let's start looking at the "underlying Risk factors" they used to dismiss the causal link.
I will expand this article, adding links to what others have written about Stroke after the mRNA jabs, but thought it might be useful to start the ball rolling with a very interesting article I just found.1
John Hallenbeck is an expert in spinal cord–damaging Decompression Sickness, who focused on the Blood–Endothelial Interface, and his research was influenced by the local Shwartzman phenomenon which will be examined more closely in another Substack article I am planning.
Look at Hallenbeck’s Figure where he shows Paralysed or Dead among different types of Rats he used in experiments where he jabbed them with Intracisternal doses of Endotoxin.
First note that Sprague-Dawley (SD) Rats were much more susceptible than Wistar Rats, the latter being favourite rats used in BioNTech/Pfizer jab experiments.
Then note in increasing percent Paralysed or Killed we see Diabetic Sprague-Dawley (SDDIB), 24-month old Sprague-Dawley (SD2Y), Spontaneously Hypertensive (SHR), Stroke Prone Spontaneously Hypertensive (SHRSP) and finally Aged Spontaneously Hypertensive Retired Breeder (SHRRB).
I think we can relate this progression to Human Jab Stroke victims and perhaps interested Data Deep Divers might like to see if we can correlate reported cases in VAERS or other databases.
Pfizer mRNA Jab Stroke Victims
Pfizer reported thousands of cases of Stroke after its mRNA under more than 22 different headings in its report.2
These included the following case numbers to April 2022:
Ischaemic stroke 1,339; Cerebral haemorrhage 1,105; Subarachnoid haemorrhage 378; Haemorrhagic stroke 173; Embolic stroke 135; Haemorrhage intracranial 95; Cerebellar stroke 60; Cerebellar haemorrhage 60; Thalamus haemorrhage 45; Brain Stem haemorrhage 44; Thrombotic stroke 36; Lacunar stroke 35; Subdural haemorrhage 34; Putamen haemorrhage 33; Basal ganglia haemorrhage 30; Brain stem stroke 25; Haemorrhagic transformation stroke 25; Vertebrobasilar stroke 17; Cerebral microhaemorrhage 16; Traumatic intracranial haemorrhage 11; Basal ganglia stroke 9; Stroke in evolution 8; Spinal cord haemorrhage 4; Haemorrhage Neonatal 3; Spinal stroke 3.
I am also very interested in Pfizer victims reporting:
Cerebral Haemorrhage Foetal 4; Disseminated Intravascular Coagulation in Newborn 2; Cerebral Haemorrhage Neonatal 2; Disseminated Intravascular Coagulation 108; Pituitary Haemorrhage 2
Stroke after Jabs, where to now?
Discussion is open.
Hallenbeck J. 2010. How inflammation modulates central nervous system vessel activation and provides targets for intervention—a personal perspective. https://nyaspubs.onlinelibrary.wiley.com/doi/10.1111/j.1749-6632.2010.05785.x
APPENDIX 2.1 Cumulative Number of Case Reports (Serious and Non-Serious, Medically Confirmed and Non Medically-Confirmed) from Post-Marketing Data Sources, Overall, by Sex, Country, Age Groups and in Special Populations and Summary Tabulation by Preferred Term and MedDRA System Organ Class BNT162B2 - ALL FOI-3727-01 https://www.tga.gov.au/foi-disclosure-log
Inflammation is a complex process that involves the activation of immune cells in response to tissue injury or infection.
In the central nervous system (CNS), inflammation can occur in response to a variety of stimuli, including infection, trauma, and neurodegenerative diseases.
One of the key components of CNS inflammation is the activation of blood vessels in the brain. This activation is known as neurovascular coupling and is thought to play an important role in regulating blood flow to regions of the brain that are active during cognitive or sensory tasks.
Inflammation can modulate neurovascular coupling by altering the response of blood vessels to neuronal activity.
This can lead to changes in cerebral blood flow, which can have important implications for brain function.
In addition to its effects on neurovascular coupling, inflammation can also contribute to the development of various CNS diseases, such as stroke, multiple sclerosis, & Alzheimer's disease.
In these conditions, inflammation can lead to damage to blood vessels and neurons, which can result in cognitive and motor deficits.
Interventions that target inflammation in the CNS have the potential to improve outcomes in these diseases.
For example, drugs that inhibit the activity of immune cells or reduce inflammation in the brain have shown promise in preclinical studies and clinical trials.
Inflammation plays a critical role in modulating CNS vessel activation & is a promising target for intervention in a range of neurological diseases...