CD56 and CD57 Natural Killer Cells in Jab Myocarditis - Endotoxin Effects

A recent paper on Jab induced Myocarditis helps explain why Males are more affected but the authors seem to be unaware that Endotoxin affects the degree of Killer Cell differentiation and maturation.

The paper in question¹ was brought to my attention by one of my subscribers Dr Robert W. Chandler.

The Myocarditis paper was written by Jab enthusiasts who claim:

The effectiveness of the COVID-19 vaccines was demonstrated by preventing 89.1% of COVID-19-related hospitalizations, 97.4% of COVID-19-related admissions to an intensive care unit, and 99.0% of COVID-19-related deaths.

Here is their graphical abstract:

The abbreviations used are NK = Natural Killer; IL = Interleukin; CD = Cluster of Differentiation; IFN = Interferon; TNF = Tumor Necrosis Factor; KIR = Killer cell Immunoglobulin-like Receptor; DNAM-1 = DNAX accessory molecule-1 (also known as CD226 molecule²); TnT = Troponin T.

The study was done exclusively on Han Chinese people which might affect their findings of some Genetic risk associations.

NK cells exacerbate the development of systemic inflammation by production of cytokines that regulate the activity of Macrophages, Dendritic cells, and Neutrophils. Under normal circumstances activated NK cells also exert an inhibitory effect on the immune system by production of immunosuppressive cytokines such as IL-10.

In the rest of this article I discuss the fact that the authors, who are based in Hong Kong, Macau and UK, make no reference at all to the known effects of Endotoxin on the CD56 and CD57 cells.